Abstract
Several studies suggested that platelets might play a crucial
role in #pathogenesis of inflammatory #boweldisease (IBD) [1,2].
There is solid evidence demonstrating that platelets, in addition
to their traditional role in #hemostasis, can also function as potent
#proinflammatorycells [3]. Upon activation, platelets secrete a
large number of biologically active molecules able to activate an
inflammatory process. At the same time, the morphology of platelets
changes and their mean volume (MPV) reduces [4]. Low MPV can
be a marker of activity of IBD [5]. However, the way the functional
properties of #platelets change, primarily their #aggregation capacity,
remains unclear. Small platelets have lower functional abilities and
larger platelets are metabolically and #enzymatically more active
[6,7].
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