Abstract
The constitutive susceptibilities to malignant transformation are
well illustrated by the evolving dimensions of a dysfunctional #ubiquitin proteasomal participation in generative reformulation of #carcinogenetic pathways of interactivity. Further considerations indicate the
transformation as derivative dysfunctional ubiquitin proteasomal system
for further transformation. The multilayered complexities of such
changes attest for redistribution of protein intermediates within
various cellular #subcompartments as signified by #endosomal and nuclear
systems of cooperative interchange. Dynamics formulation is itself a
parametric measurement in substitution modelling of substrates as indeed
projected within system profile for further change.
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